EMF Block Paint plus EMF Shielding
Whether you live in an apartment or a house, or you just would like to keep your home free of EMFs There are plenty of ways you can reduce exposure. One of the easiest is to reduce your electronic device use. You can also turn to EMF blocker paint to block EMF radiation from entering your home. Another method to protect your house from EMF radiation would be to put up a RF shielding canopy. This is a cloth made of net which contains EMF shielding. It's used to prevent EMFs from entering a room. https://mcnulty-glass.blogbright.net/info-about-emf-blocking-radiation-1681268849 is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.
Several studies have shown studies have shown that the nonionizing EMF produces antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation in cancer-related stem cells. This could explain the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's effects on patients with cancer is not clear.
Aspects from AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumours were divided into 3 groups. First, the group that was unaffected RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to that used in humans. In the third, they were subjected to RF EMF with HCC-specific modulation frequencies. emf blocking of HCCMF on the tumours was evaluated against that of RCF. The results showed that the tumours treated with HCCMF were significantly shrinking. However, tumours treated with RCF showed no evidence of shrinkage in the tumour.
The mechanism of tumor-specific AM RF EMF may be due to the fact that cancer cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. emf blocker on HCC cells is controlled by CACNA1H the protein that mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H may have broader implications for the treatment and diagnosis of various cancers.
The tumors in the controls were never exposed to RF EMF, and were fed a standard mouse diet. The tumors of HCCMF HCCMF group were treated with Huh7 cells when they were 5 to 7 weeks old. The tumors were then killed after they had a high burden.
The tumors of the three groups also showed different growth curves. The tumors treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, tumors that were treated using RCF showed no reduction in size. The difference was highly significant. The tumors treated by RCF showed necrosis that is common when tumors are that are exposed to RCF. It is possible that the necrosis was caused by the lack of oxygen in the more invasive cancers.
In conclusion, the findings show an AM-RF EMF has anticancer effects in vitro and in vivo. A number of studies have proven the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained effect on the development of HCC tumours in living tissue.