EMF Block Paint in addition to EMF Shielding
If you reside in a house or apartment or just need to ensure that your house is free of EMFs There are a variety options to limit your exposure. One of the most straightforward is to limit the use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from entering your home. Another way to shield your house against EMF radiation would be to put up an RF shielding canopy. It is a type of net that contains EMF shielding. It's used to stop EMFs from entering rooms. Another option is to get your home fitted with an electrical enclosure. They are referred to as Faraday cages.
Several studies have shown how the EMF that is not ionized produces anti-proliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to involve down-regulation in cancer-related stem cells. emf blocking may account for the long-term effects observed in certain patients suffering from advanced HCC. But, the reason for AM EMF's effects on cancer patients is not clear.
Effects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumors were classified in three different groups. The first group was not exposed to RF EMF. Another group of participants was subjected to RF EMF at a frequency that is similar to the frequency used by humans. Third group members were exposed to RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on tumours was assessed against the effect of RCF. The results showed that the tumors treated by HCCMF showed significant shrinkage. However, the tumours treated with RCF didn't show evidence of shrinkage in the tumour.
The reason for tumor-specific AM RF EMF could be driven by the fact that tumor cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. https://wang-cheng.blogbright.net/details-of-emf-blocking-radiation-1681268633 to inhibit proliferation upon HCC cells is controlled through CACNA1H which is a protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications in the diagnosis and treatment of a variety of cancers.
The tumors in the control group were not exposed to EMF from radiofrequency, and fed a normal mouse diet. The tumours in HCCMF HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumors were then killed in cases of excessive burden.
The tumours in the three groups also displayed different growth curves. The tumours treated with HCCMF had a significant reduction in the size of the tumour after 8 weeks. However, the tumors that were treated using RCF didn't show signs of shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis that is common in tumours exposed to RCF. It is possible that the necrosis was caused by a lack of oxygen in the larger tumors.
In sum, the results suggest an AM-RF EMF has anticancer activity in vitro and in live. Several studies have shown that AM RF EMF produces measurable reduction in tumours within HCC patients. There is a possibility that AM RF EMF triggers these effects through CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting impact on the growth of HCC tumors in vivo.